To investigate the effect of atorvastatin on gap junction remodeling in hypertrophied myocardium induced by pressure-overload.
目的探讨阿托伐他汀对心肌肥厚时缝隙连接重构的影响。
Hypertrophied myocardium is prone to proarrhythmic toxicity and development of the torsade DE points type of ventricular tachycardia.
肥厚心肌对于抗心律失常药物的致心律失常毒性非常敏感,容易发生尖端扭转型室速。